Transcript

[00:00:00] Today’s episode is brought to you by DaxCopilot from Microsoft. DaxCopilot is an AI assistant that helps automate clinical documentation and workflow to let you be more efficient and reduce all the administrative burden that comes with patient care. To learn more about how DaxCopilot can help improve healthcare experiences for both you and your patients, visit aka.

ms slash knock, knock, hi. Again, that’s aka. ms. Dot M S slash knock, knock. Hi.

Hello, everybody. Welcome to a jet lagged edition of knock, knock. I I’m your host, Dr. Glaucon Flecken. I really shouldn’t say jet lag. That implies like. Moving across a huge number of [00:01:00] time zones, uh, more just like tired. I learned something about myself. I, I, I ran, I’m a private practice ophthalmologist as you all know.

Research is not my forte, but I, I did a little experiment. I did. Uh, I wanted to see how well I could function after a red eye flight. So here’s why. Because, uh, in general, I don’t like traveling to the East coast. I wouldn’t say East Coast. Eastern time zone. I don’t, that is by far, all due respect to all of you listening who live in the Eastern time zone.

Your time zone sucks. It’s, it is the worst, it’s the worst time zone. Give me, honestly, give me one good thing about the Eastern time zone. I would love to hear it. All your favorite TV shows, not, I mean, now it’s everything streaming, so that’s, that ages me, saying that, uh, sporting [00:02:00] events all come on later, uh, just everything, like, uh, media wise comes up later, uh, it’s, um, in traveling to that time zone, I really, I think I just gave you just one example.

But, honestly, that’s a big one, especially when I like football, and you have to wait so late, and you’re up so late watching like Sunday Night Football, Monday Night Football, you know, I don’t like it. Anyway, but the even worse part about it is, is traveling from the west coast to the east coast. It’s, it takes a whole day.

I have to take an entire day off from work just to travel, to like, give a talk in like, you know, New York or Philadelphia or whatever. And uh, and it just, it’s hard because I can only take off so many days. Occasionally, I do have to see patients in clinic. I do try to practice ophthalmology from time to time.

And so, uh, uh, just, uh, yesterday. Yesterday, I took a trip, I took a [00:03:00] red eye, actually the red eye left Monday night, so I worked Monday, all day, and I left at 11pm from Portland, and I arrived in Detroit, uh, uh, you know, 5 hours later. 6:00 AM and then I spoke at a, at a symposium. So I was up most of the day and I don’t sleep on planes.

And the, the, the experiment was, can I do this? That’s the, the, like, I, I’ve always been nervous to do it. I’ve wanted to do it because I feel like it’s a wasted day, uh, because just traveling. And so I say if I can just, if I can fly on a red eye and be like. funny enough on no sleep or very little sleep, then, uh, then I can do this.

And, and, and, and turns out I can. So Kristen and I, we both went, Kristen though, she, she does not have an ophthalmology clinic to do. Um, and so she has a much more flexible schedule than me. So she went early. She was like, Forget that, there’s [00:04:00] no way in hell I’m flying with you on a red eye. She came earlier and we spoke together at the Mary Tyler Moore Vision Initiative Symposium.

This is a new thing that we are ambassadors to, uh, it’s a, it’s a non profit, uh, research Outreach based initiative for patients with diabetic retinopathy who have vision loss from diabetic retinopathy. And um, it’s a really cool organization that was, the whole thing was founded by Mary Tyler Moore who died a number of years ago.

Her husband, Robert Levine, Dr. Robert Levine, he’s a, he’s a physician. Started this because Mary Tyler Moore had diabetes and she’s, um, in particular, she had vision loss related to diabetic retinopathy and one of the things that she wanted was to try to end this problem, end this [00:05:00] disease, uh, at least end the, the vision disability that comes with severe diabetic retinopathy and so he’s done over the years, put all these, all this work together to bring in, uh, The latest research, they have a partnership with the Kellogg, um, uh, department of Ophthalmology at University of Michigan.

So all these amazing researchers that are doing things in AI and pharmaceuticals and devices and, and, uh, things to, to try to, to improve the, the diagnosis and treatment of diabetic retinopathy to make it just less of a terrible disease that causes blindness. Just trying to move the field forward because.

There’s really, I mean, there’s been some, some, some, some good things that have come over the last few decades, but uh, not as much as you would think for such a common disease. I mean, we’re talking, you know, millions and millions of people have vision loss because of diabetes. And so, [00:06:00] we just wanted to be a part of it.

And so, you know, we, uh, Kristen and I are ambassadors. Our job It’s to get the word out because it’s so interesting being a, it was intimidating, honestly, being at a place like this with so many people who are talking about very high level things, uh, and they’re like at the forefront of their field, uh, world renowned researchers, physician scientists.

And then I get up on stage and I’m like, I make TikToks, that’s what I do, and Kristen here, she, uh, is, has, you know, she runs the TikTok making business, uh, and honestly does a lot of the work anyway, uh, of just, you know, actually making this into a thing, and, uh, and so anyway. We were there, we’re there, we told our story and, um, uh, just talked about social media advocacy and how we can help get the word out because, you know, researchers are good at [00:07:00] research, maybe not so good at trying to get, you know, some of these amazing things they’re doing out to the public in a way that’s accessible and that people can understand.

So we talked about that, it was great, great experience, even though I was doing it after a red eye. We, I still was able to tell funny jokes. I felt like my energy was pretty good. Maybe not what it usually is. It’s pretty good. Uh, and, and then I got home and just, and crashed and then worked today. And I’ll tell you the day after like two days after the red eye, that’s when it hits you, like I flew all night and then was up the next day I was doing okay.

And then the next day after that, that’s when it’s like, Whoa, That was a mistake, what I just did. So anyway, thanks Eastern Time Zone for making me do this. It’s your fault. It’s your fault. The worst time zone, by far. I honestly, I think Central is, is Actually, I think [00:08:00] Mountain’s probably the best, and then Central, then Pacific, then, then Eastern.

Yeah, those are the final rankings and no one can argue with me. Uh, okay, what else? Um, well, I guess, since I brought up diabetic retinopathy, I guess I just talk a little bit more about that. I did have a, uh, you guys should look back. I did an episode about diabetic retinopathy and, uh, you know, So, one question this might bring up for people is what, what actually, what is the cause of the vision loss?

You talk about blindness from diabetic retinopathy, what does that look like? And usually what that means, so, because diabetes is a disease that affects the small blood vessels. You basically get destruction of blood vessels in your body. It’s a vascular disease. So in your heart, in your kidneys, A lot of organs throughout your body, your, your feet, your hands, you know, you start losing some of the small, those little capillaries that bring blood flow and then, and then it, [00:09:00] so it causes damage to your organs.

Well, the eyes are no different. The eyes are special with, with diabetes though because we can, we can actually visualize. It’s the easiest way to visualize the actual damage. I can look at someone’s retina with diabetic retinopathy and I see. The capillaries, we see the small little arterials that start developing little micro aneurysms that start swelling and bleeding and leaking out into the retina.

We can get a cross sectional image of the macula that shows all the seven or eight or I forget how many layers there are in your retina and we can see the swelling, the fluid leaking from the small, those capillaries and causing separation between the layers of the retina. Resulting in vision loss for the patient.

It’s remarkable, some of the things we can do. And it’s just getting better with things like this Mary Tyler Moore Vision Initiative. And so, you know, as the diabetic retinopathy gets [00:10:00] worse, the blood vessels become, you know, more tenuous and leaking more, and there’s more destruction there. And then, when you’re not having adequate blood flow to the retina, The body’s like, oh shit, we gotta get more blood flow.

How are we gonna do this? Let’s make new blood vessels. Here’s this chemical. Vascular endothelial growth factor. We know this will make blood vessels. Let’s make a bunch of blood vessels as quickly as possible. Well, the body does it, and it’s really good at it. That VEGF is powerful, but those blood vessels, they’re not good.

It’s like trying to build a house, like, in a day. Like, unless you’re Amish, it’s not going to work out too well. And so, it’s the blood vessels that the body, the eye makes, are not good. are wispy and prone to leaking and prone to bleeding. So, we heard stories at this symposium from patients about, um, about what it’s [00:11:00] like to like have a bleed.

You’re just, you’re going about your business, you’re at work, or you’re on vacation and all of a sudden you lose vision. And sure enough you have a bleed in the eye. Those, one of those blood vessels broke open. Eye filled with blood, it can take months for that to recover, for the eye to clear that blood out, eventually it does, sometimes it doesn’t, you have to have surgery, a vitrectomy to remove that blood, and then eventually if that, those blood vessels, if it gets bad enough, you get all these abnormal blood vessels, you start to get scarring, and that can result in retinal detachments, and now we’re getting stuck in end stage diabetic retinopathy, and we’re trying to avoid that.

Alright, diabetes, that’s a disease that’s not going away, alright, so at least not now until maybe we have gene therapy or something down the road that can totally just get rid of diabetes. But if we’re going to have diabetes as a disease, then we need [00:12:00] to do everything we can and work toward preventing the devastating visual complications that can occur with diabetic retinopathy.

And that includes getting people diagnosed sooner, getting education to patients so they know what the, it’s the saddest thing are the patients who were in their twenties and thirties and have severe vision loss. And there’s a lot of factors that go into that. Having access to medical care, having access to insulin, and this is a multi factorial thing and so on the ophthalmology side, on the diagnostics, the treatment side, we’re working, trying to figure out, doing what we can.

And I really, I was just like, I left this, the Mary Tyler Moore symposium, I just energized. Try to defeat diabetic retinopathy. All right. So, uh, anyway, I’m just real excited to be a part of it. Not excited for future visits to the East coast, but that’s okay. I can get over that for a good cause. All right.

Let’s take a break.[00:13:00] 

Kristen, we got to talk about disability insurance. Why’s that? Because I don’t have it! I know. I wish I did. I wish you did too. I should have gotten it before I had testicular cancer, and testicular cancer again, and a cardiac arrest. Yes, we thought we had time, but it turned out we did not. Well, let me tell you about Pearson Rabbits.

Tell me. This is a company founded by a physician, Dr. Stephanie Pearson, an OBGYN who had an injury, was unable to work, and is now an advocate for physicians and wants to help others avoid similar mistakes. Hmm. Hmm. Pierson Ravitz has decades of experience, takes its time to get to know you, your medical history, and what you need.

It’s not just a form you fill out on the website. Yeah. That’s a personal connection they make and they’ve helped more than 6, 000 physicians get disability insurance. Hmm. I wonder if they can help you. Maybe. To find out more and get a free one to one consultation, go to www. PiersonRavitz. com slash knock knock.

Again, that’s [00:14:00] Pearson Rabbits, P E A R S O N R A V I T Z dot com slash knock knock to get more information and protect your greatest investment, you.

By the way, Kristen is not happy with my conclusion that it’s okay. I don’t, I like, I can very easily take a red eye across the country and still do stuff the next day because she made a good point. She told me, yeah. You know, you have to sleep eventually. I do forget that. Like, sure, I’m doing a red eye. I can function the next day.

Eventually I do have to sleep and I’ve gotta catch up on that sleep. But, you know, I’m not a future oriented thinker. That’s not how I operate. I just, I’m day to day. That’s, you know, which is just how I, how I go about things. Okay. So, uh, I went through some, let’s do some YouTube comments. We got some good ones.

So I [00:15:00] just recently. This is of the most recently posted video which is, does your eye color make you prone to certain diseases? We talked about a number of things on that episode. One of them was pigmentary glaucoma, that was the main topic. And uh, and so this is um, by the way, all these, these, these videos, the episodes are up on our YouTube channel at Glaucomfleckens.

Check it out. We’d love seeing those comments. If you want me to read your comments, some people are like, Oh, I’m so glad you read the comments. I love reading the comments. I love reading what you guys are writing to me, and you’re giving me great suggestions, great topics to cover. All right, so I’m gonna go through a few of these comments.

This is, this made me laugh. So, Pigmentary Glaucoma, again, just two sentences. When you have brown eyes and sometimes you can release pigment. You have a lot [00:16:00] of pigment in your iris. You release pigment that clogs up the tear, the drainage system inside the eye, the trabecular meshwork, and can cause pressure spikes.

And these typically happen in the classic demographic is young adult males who are slightly myopic. And I got a comment from at Rowan Jacobs who says, As a brown eyed myopic man in my 30s, this was a harrowing listen. Ah, that made me laugh, thank you, but don’t, don’t be, don’t be nervous Rowan and anybody else who falls into that group, alright?

It’s still a very rare, but if you, if you do, especially after vigorous activity, if you notice eye pain, you might want to get it checked out. Go to your, go to your eye doctor. Okay, here’s a, here’s a comment from at Amanda. Be Evan, 6331 said, Hello, I am curious about issues that arise from having [00:17:00] astigmatism in everyday life, and if it is true that there is really nothing to do about it.

That’s totally false. There’s lots of things to do about astigmatism, but the comment keeps going. It says, More specifically, as our daylight dwindles and night driving becomes necessary, the glare and flare that occurs around every source of light becomes more of an issue. With trends now of headlights switching to LEDs and more of the bright blue, not warm yellow, I find those flares are larger and make driving much more difficult.

Thank goodness for fog lines to help me stay on course when a large truck or SUV passes me going in the other direction. I am so glad you brought this up because most of my patients do as well. So there’s a lot to dive into with this comment. Let’s talk about astigmatism for a second. So the first thing to know is that But almost, pretty much everybody has some degree of astigmatism.[00:18:00] 

Astigmatism just describes the shape of your cornea. Alright? If you have astigmatism, your cornea, it’s shaped a little bit more like a football, like an American football, as opposed to like a basketball. Nobody’s cornea is perfectly spherical. Nobody’s cornea. That’s, that would be Extremely rare. The question is, how much astigmatism do you have?

If it reaches a certain threshold of astigmatism, you start to have symptoms. It starts to be visually significant. And what, uh, Amanda’s talking about here is, uh, these flares of light around the eyes. Sources of light, these flares around sources of light. So I use at night in the winter, I use Christmas lights as an example for people, pinpoint light, and if you have a lot of astigmatism, it’ll look like you have these, like a trail, like a comet tail of light coming off in different directions, depending on where, what kind of astigmatism you have.

So [00:19:00] it’s like all these lights are stretched out. They look very unusual and it can be kind of pretty, but probably not when you’re trying to actually drive or do, you know. Uh, I’ve had people tell me that they love taking their glasses off and just looking at Christmas lights, uh, because, because the, you get all this, the sparkles and the flares and it just, it’s very colorful, lots of light.

Um, but again, if you’re trying to function, it’s not a great thing. And um, and so what can you do about astigmatism? Well, obviously glasses and contacts, that’s like the basic thing. And that’s great for regular astigmatism. Alright, that’s your just normal, average amount of astigmatism. But a lot of people have irregular astigmatism.

Things like keratoconus, pellucid, mar marginal degen degeneration, other corneal ectasia where you have a cornea disease. Astigmatism is not a disease. Right. It’s, it’s, it’s a, it’s just a, a normal, um, [00:20:00] finding, like myopia. It’s like myopia, astigmatism, you know, these are things we can correct with glasses.

But a a, uh, pathologic state for the cornea would be things like keratoconus, where you have a degeneration, right? Ectasia of the cornea. Uh, also severe dry eye can induce a lot of astigmatism. So for these things, you, you treat the underlying disease. For keratoconus, and we had an episode on that a while back as well, we have this thing called corneal collagen crosslinking, where you, really what you do is you take the top off of the cornea, the epithelium, you rub off all the epithelial cells, and then you bathe the cornea in these riboflavin eyedrops, riboflavin 6 phosphate.

You just bathe it like the cornea just gets full, engorged. It’s corneal engorgement with riboflavin and then we shine ultraviolet light on the cornea and what it does is it [00:21:00] stiffens. Stiffens up the cornea. We do that in people with rapidly progressing keratoconus who you don’t want it to keep progressing and so you do this treatment and it stiffens up the cornea and so it won’t progress as much.

Because keratoconus is like a weakening of the cornea. So yeah, there’s treatments, there’s lots of things we can do for, for, and then once you have cataract surgery, well what you can do then is you take the lens out, and the artificial lens we put in there, that has astigmatism correction in it, if you want it to be.

There’s special lenses that do that. It’s basically like taking your astigmatism correcting glasses, taking that lens and just shrinking it down, changing the shape a little bit, and sticking it inside the eye, so it’s there permanently. So lots of things to do about astigmatism. But making sure you know it’s regular, normal kind of astigmatism, or if it’s not, how to treat it.

But, I really appreciate you bringing up, Amanda, the second part of your [00:22:00] comment about the headlights. This is like These are easily top three complaints that I hear from patients all the time. What is going on with the headlights? What is happening? They’re getting worse. And so I thought at first, when I first started practicing ophthalmology, I was like, okay, the headlight thing, this, it’s a, it’s a cataract symptom.

People start having problems with headlights and we ask about it, honestly. We ask about the headlights. Do you have issues with glare, with oncoming traffic? And, and then people just go, Oh, Doc, those, I hate those blue headlights. I hate them. And so this is a big thing with, with cataracts and cataract surgery typically helps that.

And at the beginning of my career, I was like, okay, this is just a cataract thing. Like, this is just what happens as you get older. But now, over the past few years, even, I don’t have any cataract. Most people under the [00:23:00] age of 50 don’t have cataract. I’m getting people of all ages starting to complain about the headlights.

They’re too bright. And I think it’s no longer, it’s, it’s, it’s now getting to the point where it’s not just cataracts. It’s just the normal human eye is not Not willing or able to tolerate the intensity of light coming from cars. It’s a thing. And so I did some research to see if there’s any regulation in it.

And there is a level where it’s like, you can’t, it’s not street legal, you know, it’s not like against the law, but would you be able to get a registration? Maybe not. Inspection. Do they check headlight intensity? I don’t know, but there are guidelines. That’s mostly what I was finding is guidelines, and the guidelines say you need to have the, the number of lumens from your light less than 3, 000.

So just to give you a frame of [00:24:00] reference, a lumen is a measure of the intensity of light, I believe. The physics people, you can correct me on that if I’m wrong, but your average halogen light bulb that fills a room is like a thousand, a thousand lumens. And so it seems reasonable, like 3000, yeah, it’s, you want it, you want to have the light on your headlight be brighter than what you have in your room.

Sure. So 3000. That’s the number I kept being thrown out there. Like okay, like the general like transportation association, whatever guidelines, like no brighter than 3000. Well now, a lot of cars don’t have halogen type lights. They have LED lights. And these LED lights. You can find them for like, like 12, 000 lumens.

And so they routinely are like 4, 000, 5, 000, 6, 000, 12, 000 lumens. It’s like staring at the surface of the [00:25:00] sun and, and. The, and the thing is, the reason I, this is a, apparently a much worse problem in the US compared to European countries or other countries around the world because we are way behind with regulating this now.

So in, in, in European countries, they have, um, these, uh, adaptive headlights. This is like a huge deal. Adaptive headlights that. What they can do, they’re like smart headlights. And what they can do is they have sensors that will automatically change the shape. Brightness and direction of the light on your car, on your headlight, to both help with making sure you maintain the driver’s visibility, but also not blinding other cars coming the opposite direction.

And this is becoming very, very common, almost standard for [00:26:00] European vehicles. It’s taking time, but it’s happening. Uh, but, uh, The U. S. is, because, you’re dragging our feet, because I don’t know if you’ve been able to tell, like, it’s, you can’t force anybody to do anything in this country. It’s not like there could be a mandate.

Let’s have adaptive driving beam headlights on our cars, on all of our cars, so we don’t blind our neighbors. It’s not, not that easy. Not that easy. So I don’t know. I don’t, I don’t think it’s a problem. It’s going to end up like the, it really does. It’s very interesting. Like on my car, granted I haven’t bought a car in seven years, but the car, the cars are just like massive computers.

It’s like driving a spaceship. We have a minivan. It’s high tech, high tech minivan. Um, But as high tech as these things like really are, are the function of our headlights, it’s just like click and then click for brighter and then [00:27:00] click for brightest. Like that’s all it does. You would think that with all the technology we have, like we’d have something better.

Well, that does exist, just mostly not in this country. All that to say Sorry Amanda, uh, I, I, I sympathize with you about the lights. So do like almost every single one of my patients and um, I don’t think we’re very good at this country of, of making that better. That would be a good platform for me to run, run on for president.

I’m going to have a platform of getting rid of the headlights. Getting rid of LED headlights, getting rid of Vizine, uh, outlawing the mandatory jail sentence if you sleep in your contacts. What else? What else is on my agenda? Um, Vizine, you can’t ever say anything bad about somebody’s glasses, unless you’re me.

I don’t know, I’ll think, no, oh, [00:28:00] eyeball tattoos, uh, you can’t do that, common sense things. Alright, this is my platform. Okay, one more break.

Alright, so I have, let’s see, one other good question that I got was from at Steven Just 01 said, I wonder if there’s any long term complications of nystagmus. I was born with it, so I don’t notice the motion, but I’m wondering if there’s any long term things to watch out for. I just imagine comparing the lens of my eye to a building in an earthquake that never ends.

Will the fibers holding it in place snap from stress fatigue? I love this comment, there’s so much to unpack here. So, first of all So, it’s basically a question, I want, is there any long term complications of nystagmus? So let me just remind you all what nystagmus [00:29:00] is. Nystagmus is an abnormal movement of the eye.

There’s a lot of different reasons that someone can have nystagmus. It can be congenital, it can be acquired. Congenital nystagmus It doesn’t necessarily portend some horrible disease, vision. You can have this latent nystagmus, which is a normal kind of movement of the eye, much more common when you cover one eye and you just have a single eye.

So the eye will kind of flick quickly and then slowly move and then flick and then slowly move back, flick, slowly move back, and it can be very rapid, it can be very slow. Uh, it’s always, I’d say nystagmus is always something that should be evaluated, even if it’s congenital and it doesn’t, there’s no problem there, it should always be evaluated because you want to make sure that it doesn’t, it’s not something like a brain tumor or a stroke or something else, uh, which can, things that can [00:30:00] cause a nystagmus from developing, so more acquired.

causes of nystagmus. Uh, things like, like, uh, uh, aniridia or basically diseases that can cause bilateral severe vision loss at a very early age. You end up with like a congenital type nystagmus. Um, so in, in, in patients with aniridia, sometimes they have Hypoplasia of their macula, so their retina doesn’t develop normally, you don’t have that foveal dip, uh, and so the, um, the macula doesn’t develop, vision doesn’t develop normally, they’re like 2200, 2400, they can have some nystagmus.

It’s kind of like a, a way for the eyes, they’re kind of like searching, eyes are kind of searching a little bit. And so, for people like Steven here, who said he was born with it, so I don’t notice the motions. What is he talking about? You would think that someone with nystagmus, no matter who you are, the eyes are [00:31:00] constantly moving.

Like your, your world is going to be constantly moving. You’re, you’re, you’re going to see something. But not always. Not always. In fact, most of the time people don’t. That’s a symptom called osillopsia. Osillopsia is you have an abnormal eye movement that results in blindness. Perception that the world is spinning or moving or shaking, it’s symptomatic.

So a lot of times we’ll see that in people with an acquired version of nystagmus. There’s some pathologic process going on, the eyes are moving funny. And they’re noticing it. Someone with latent nystagmus or congenital nystagmus, they’re not going to have that sensation. Their brain has just learned to accommodate that abnormal movement.

So they’re not constantly seeing that the world is moving. And then he questions, uh, if there’s any long term things to watch out for. So this is a really good question. And actually one that I, I probably, I think I [00:32:00] had in residency as well. It’s like this eye is constantly moving, could there could there be damage inside the eye from that?

And what he’s asking about are the fibers that holding the lens in place. So if you recall from our many anatomy discussions that we’ve had over the months, the lens inside the eye is suspended like a suspension bridge. It’s suspended by these little strings that are called zonules. They go all around the equator of the lens, holding that lens in place.

Well, there are certain diseases where those zonule fibers can be damaged, obviously in any trauma, or they could just be, uh, you can lose some of them through the disease process. Um, the, uh, and so the concern that nystagmus can just eventually cause those things to break down, that doesn’t happen. Just think about how many eye movements you have in a day.

[00:33:00] Thousands. We call them saccades. So if you look from like one finger to the other, you’re generating, those are voluntary saccades. Alright, whenever you try to scan across the room, your eyes, unless you’re following something that’s moving, your eyes don’t, they’re not, it’s not a smooth pursuit. It’s if I’m trying to scan across, I have tiny movements all the way across.

Those are little saccades. And so you have saccades with nystagmus as well. They’re just very tiny. And so, regardless of whether or not you have nystagmus, we’re constantly moving our eyes all around and it doesn’t cause damage to the eye. That’s what the eye’s for. That’s what it’s supposed to be doing.

So, don’t worry about that. The only way to have damage from those zonules holding the lens in place is from some other disease or trauma or something. But I do appreciate the question. That’s a really good one. I don’t think I saw anything, any other questions that we can get to today. Bye. Bye. I mentioned how much I appreciated the eyeball statue [00:34:00] in Dallas as the like, uh, the place of pilgrimage for all ophthalmologists.

They have this like 30 foot tall eyeball. And then, um, somebody told me that there are eyeball sculptures on the campus of Williams College in Williamstown, Massachusetts. I have been there. I’ve been to Williams College. I played, uh, I played ultimate Frisbee for the Dartmouth team. Um, and, and, uh. And we went to Williams.

They kicked our ass. They were really good that year. But yes, I’ve been to Williams. It was a really pretty place. That was at Sarah G was 2 4, 9 4. Thank you for that comment. I guess we’ll just, we’ll end it there. I think that’s enough eyeball stuff for today. Alright, let me know what you wanna talk about.

Lemme know if you have any ideas or, um, or, uh, you know, if you have any, if you have a disease you want me to talk about, uh, or a family member has some kind of eye problem. Or just a burning question. Maybe about eyeball burning. I don’t [00:35:00] know. Whatever you want to talk about. Uh, leave a comment. You don’t have to actually watch.

If you like to just listen to me and not have to not watch me talk, maybe you’re doing chores. You just want to have me in your ear. That’s a great idea. Uh, just come back later to the YouTube video, leave a comment. That’s where I’m going to address, uh, anything you guys talk about or want me to talk about.

Obviously, any, uh, any corrections, anything I got wrong, let me know. I’m not perfect and I’m a little bit sleep deprived right now, so it wouldn’t shock me. But that’s it. Thank you all for listening. I’m your host, Wilfred Anastas, Dr. Glaucomflecken. Thanks to our executive producers, Aron Korney, Rob Goldman, and Shahnti Brooke, editor engineers Jason Portizo, our music is by Omer Knock High is a human content production.

We’ll see you next time, [00:36:00] everyone.